Tuesday, January 14, 2014
Monday, January 13, 2014
The Enzyme Elastin Plays A Role In Joint Movement
Elastin is a protein in connective tissue that is elastic and allows many tissues in the body to resume their shape after stretching or contracting. Elastin helps skin to return to its original position when it is poked or pinched. Elastin is also an important load-bearing tissue in the bodies of vertebrates-from Wikipedia. The presence of elastin is reduced with age and this leads to the onset of joint pain and poor joint flexibility. One way to remedy this problem is to increase the trace mineral level, particularly copper, zinc and manganese. These lead to more effective production of elastin and allows the flexibility to return and the pain to ebb without medications. This is a natural way to treat not only the symptom but also the heart of the problem.
Supplementing with chelated trace minerals is the most effective way to change the nutritional profile and increase mineral metabolism. Minerals can easily be tied up in the body of dogs because of their total intake of nutrients. For instance, having a water source that is high in iron can cause an antagonistic relationship between iron and copper and your pet can become deficient to a sub-clinical degree. That is to say that there are no visible symptoms but the problem persists
To be assured that mineral supplementation is effective, the minerals need to be chelated, that is raw minerals bonded to amino acids at the molecular level. This insures effective absorption because of particle size and using the more basic forms of mineral molecules instead of a mineral compound(like copper versus copper sulfate)
Always check out ingredients and look for listings like “copper amino acid chelate” or “zinc amino acid chelate” and don’t accept ingredients like “copper oxide” or “copper sulfate” as acceptable forms of supplementation.
By using basic mineral nutrition instead of medications like glucosomine(an extract of shell fish which can carry its on reactions) you are creating a healthier environment within the dog and”building a better dog” not just medicating a symptom.
Thursday, January 9, 2014
Do Consumers Really Have A Chance?
Susan Thixton of Truth About Pet Food and Petsumer Report is at the AAFCO(American Association of Feed Control Officials) and she sent this message regarding who is at the meeting.
"I just got this from Susan Thixton today which tells us something about how the deck is stacked against consumers and their pets.
"I just got this from Susan Thixton today which tells us something about how the deck is stacked against consumers and their pets.
"In the midst of representatives stating their affiliation with ‘Mars Petcare’, ‘P&G’, ‘Cargill’, ‘Purina’, Del Monte’, ‘Hills Science Diet’ and all the others in attendance – there were two consumer advocates…myself and Mollie Morrissette (weather permitting Dr. Cathy Alinovi will join us tomorrow). There are an estimated 200 representatives of industry representatives here – either directly working for a manufacturer or ingredient supplier or industry trade groups. Standing and stating my name and “Association for Truth in Pet Food” is quite an honor (kinda fun actually stating those ‘truth in pet food’ words knowing many are cringing at the name)."
Thank you Susan for your powerful voice
Wednesday, January 8, 2014
Looking Back 80 Years Regarding Hip Dysplasia
Going back in history, it was pre-1930′s to find a time when the subject of “hip dysplasia” was not mentioned in any form. Those were the times when people went to the butcher shop and the corner market and got some meat scraps and fed what was left at the dinner table from the three meals a day that the matriarch of the family collected at the end of each meal.
The depression and ensuing years brought about the change of the industry and the change of the products that came out of the family pet world. There evolved an industry that processed high carbohydrate grains, like corn, into pet foods that produced an unintended consequence of making cheaper, more easily accessible, pet foods but of much lower quality.
The unfortunate by product of using high carbohydrate corn(2,200 carbohydrate calories per pound) and building a non-meat ration resulted in a weakening of the femur and the acetablumen(the three bones that intricately fit together to form the cup for the end of the femur to fit into). This brought about the emergence of CHD(canine hip dysplasia) Now, when the malady arose, a lot of veterinarian and researchers tried to show that this was a genetic consequence.
From the 1930′s until the 1960′s, the breed associations in the United States and Europe tried their best to use genetic selection to aleviate the problem, to no avail. They found CHD problems in 70 to 75% of large breed dogs and after those three decades of work, they felt all the work had reduced the incidence by almost 5%. Not much to hang your hat on.
It was in the sixties that two men, one a veterinarian and one a scientific journalist began to explore nutrition as a major cause of CHD.
It was amazing the controversy that resulted from their findings which many years ago led to law suits and even some criminal complaints but the facts are the facts. These two researchers found that the use of high carbohydrate grains, especially corn, resulted in muscle and skeletal weaknesses that were the result of softening of the tissue and the weakening of the joints and now with some larger breeds, three-fourths of all those dogs have joint and walking problems.
NOTE: We are going to invest the time of several articles and a lot of research to further look into this problem and try to explore the economics of what this malady costs in actual dollars.
Check out THIS SITE for more blog information while waiting for part 2 of this article.
The depression and ensuing years brought about the change of the industry and the change of the products that came out of the family pet world. There evolved an industry that processed high carbohydrate grains, like corn, into pet foods that produced an unintended consequence of making cheaper, more easily accessible, pet foods but of much lower quality.
The unfortunate by product of using high carbohydrate corn(2,200 carbohydrate calories per pound) and building a non-meat ration resulted in a weakening of the femur and the acetablumen(the three bones that intricately fit together to form the cup for the end of the femur to fit into). This brought about the emergence of CHD(canine hip dysplasia) Now, when the malady arose, a lot of veterinarian and researchers tried to show that this was a genetic consequence.
From the 1930′s until the 1960′s, the breed associations in the United States and Europe tried their best to use genetic selection to aleviate the problem, to no avail. They found CHD problems in 70 to 75% of large breed dogs and after those three decades of work, they felt all the work had reduced the incidence by almost 5%. Not much to hang your hat on.
It was in the sixties that two men, one a veterinarian and one a scientific journalist began to explore nutrition as a major cause of CHD.
It was amazing the controversy that resulted from their findings which many years ago led to law suits and even some criminal complaints but the facts are the facts. These two researchers found that the use of high carbohydrate grains, especially corn, resulted in muscle and skeletal weaknesses that were the result of softening of the tissue and the weakening of the joints and now with some larger breeds, three-fourths of all those dogs have joint and walking problems.
NOTE: We are going to invest the time of several articles and a lot of research to further look into this problem and try to explore the economics of what this malady costs in actual dollars.
Check out THIS SITE for more blog information while waiting for part 2 of this article.
The Origin Of Hip Dysplasia
Going back in history, it was pre-1930's to find a time when the subject of "hip dysplasia" was not mentioned in any form. Those were the times when people went to the butcher shop and the corner market and got some meat scraps and fed what was left at the dinner table from the three meals a day that the matriarch of the family collected at the end of each meal.
The depression and ensuing years brought about the change of the industry and the change of the products that came out of the family pet world. There evolved an industry that processed high carbohydrate grains, like corn, into pet foods that produced an unintended consequence of making cheaper, more easily accessible, pet foods but of much lower quality.
The unfortunate by product of using high carbohydrate corn(2,200 carbohydrate calories per pound) and building a non-meat ration resulted in a weakening of the femur and the acetablumen(the three bones that intricately fit together to form the cup for the end of the femur to fit into). This brought about the emergence of CHP(canine hip dysplasia) Now, when the malady arose, a lot of veterinarian and researchers tried to show that this was a genetic consequence.
From the 1930's until the 1960's, the breed associations in the United States and Europe tried their best to use genetic selection to aleviate the problem, to no avail. They found CHD problems in 70 to 75% of large breed dogs and after those three decades of work, they felt all the work had reduced the incidence by almost 5%. Not much to hang your hat on.
It was in the sixties that two men, one a veterinarian and one a scientific journalist began to explore nutrition as a major cause of CHD.
It was amazing the controversy that resulted from their findings which many years ago led to law suits and even some criminal complaints but the facts are the facts. These two researchers found that the use of high carbohydrate grains, especially corn, resulted in muscle and skeletal weaknesses that were the result of softening of the tissue and the weakening of the joints and now with some larger breeds, three-fourths of all those dogs have joint and walking problems.
NOTE: We are going to invest the time of several articles and a lot of research to further look into this problem and try to explore the economics of what this malady costs in actual dollars.
The depression and ensuing years brought about the change of the industry and the change of the products that came out of the family pet world. There evolved an industry that processed high carbohydrate grains, like corn, into pet foods that produced an unintended consequence of making cheaper, more easily accessible, pet foods but of much lower quality.
The unfortunate by product of using high carbohydrate corn(2,200 carbohydrate calories per pound) and building a non-meat ration resulted in a weakening of the femur and the acetablumen(the three bones that intricately fit together to form the cup for the end of the femur to fit into). This brought about the emergence of CHP(canine hip dysplasia) Now, when the malady arose, a lot of veterinarian and researchers tried to show that this was a genetic consequence.
From the 1930's until the 1960's, the breed associations in the United States and Europe tried their best to use genetic selection to aleviate the problem, to no avail. They found CHD problems in 70 to 75% of large breed dogs and after those three decades of work, they felt all the work had reduced the incidence by almost 5%. Not much to hang your hat on.
It was in the sixties that two men, one a veterinarian and one a scientific journalist began to explore nutrition as a major cause of CHD.
It was amazing the controversy that resulted from their findings which many years ago led to law suits and even some criminal complaints but the facts are the facts. These two researchers found that the use of high carbohydrate grains, especially corn, resulted in muscle and skeletal weaknesses that were the result of softening of the tissue and the weakening of the joints and now with some larger breeds, three-fourths of all those dogs have joint and walking problems.
NOTE: We are going to invest the time of several articles and a lot of research to further look into this problem and try to explore the economics of what this malady costs in actual dollars.
Wednesday, December 18, 2013
Tuesday, December 17, 2013
What Is The FDA Smoking?
The FDA recently released the results of a study that compared raw pet food products vs. commercially prepared dry and semi-moist dog and cat foods and jerky type treats. Those results showed that they tested 480 samples of dry and semi-moist products and found 0 incidences of salmonella and 0 incidences of L. monocytogenes.
The part of the test covering jerky type treats tested 190 commercially prepared products and found 0 incidences of the same two pathogens.
Raw pet food products were also tested and of 196 formulas tested, they found 15 incidences of salmonella and 32 incidences of L. monocytogenes.
Now, the FDA proclaims that they have "zero tolerance" for incidences of salmonella and L monocytogenes so you have to wonder two things.
1.) There have been numerous recalls of dry, semi-moist pet foods and jerky type treats over the past year which occurred at the same time those recalls were going on and their testing just happened to miss all those products that have been recalled by coincidence. and
2.) They found a total of 47 incidences of pathogens in raw pet food products at an incidence rate of 24% but none of those products were targeted for recall in this "zero tolerance" world.
To not make the connection of utter nonsense, I would have to be smoking something very potent.
Note: Thanks to Susan Thixton at Truth About Pet Food for amassing and publishing this information.
The part of the test covering jerky type treats tested 190 commercially prepared products and found 0 incidences of the same two pathogens.
Raw pet food products were also tested and of 196 formulas tested, they found 15 incidences of salmonella and 32 incidences of L. monocytogenes.
Now, the FDA proclaims that they have "zero tolerance" for incidences of salmonella and L monocytogenes so you have to wonder two things.
1.) There have been numerous recalls of dry, semi-moist pet foods and jerky type treats over the past year which occurred at the same time those recalls were going on and their testing just happened to miss all those products that have been recalled by coincidence. and
2.) They found a total of 47 incidences of pathogens in raw pet food products at an incidence rate of 24% but none of those products were targeted for recall in this "zero tolerance" world.
To not make the connection of utter nonsense, I would have to be smoking something very potent.
Note: Thanks to Susan Thixton at Truth About Pet Food for amassing and publishing this information.
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